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This pool fuels essential processes such as DNA synthesis and mitochondrial respiration, but simultaneously predisposes cells to oxidative stress through iron-catalysed ROS generation (69, 70)

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Simvastatin and rosuvastatin significantly inhibited cell proliferation by suppression of RAS protein, matrix metalloproteinases (MMP-2/9) and NF-B-p65 expression in LC tissues, and this effect was dose-dependent [90]